It is not new that doctors and researchers warn about the dangers of self-medication and also the continuous use of medicine – sometimes without need. This is one of the major problems facing modern society.
With this, it was discovered that the use of some drugs can have devastating consequences, such as brain cell wear.
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According to experts from the Australian Science and Technology Organization, the use of anxiety medications can interfere with dendritic spines. They form part of a fundamental structure in neurons, which is crucial in activating brain cells.
Richard Banati, one of the authors of the research, is categorical about the conclusion of the study: there is a strong suggestion that these drugs can accelerate the development of dementia.
One of the drugs used in the research was diazepam
. In addition to anxiety, it is recommended to treat alcohol withdrawal, muscle spasms, seizures, and in some cases as a sedative before medical procedures.The researchers saw that the drug did not reach the synapses of neurons directly. However, they affected microglial cells, altering their activity and function. According to Banati, when the connections between neurons are impaired by the malfunction of these cells, it is possible for there to be a disconnection in the neural networks.
Thus, with this interference, even if subtle, a fertile environment is created for further progression of dementia. There is also the possibility of causing severe fatigue in the patient.
As stated earlier, the study leads only to a suggestion of the relationship between the continued use of these drugs and the wear and tear of brain cells. However, it is a valuable insight for the scientific community and the kickoff for further studies – these more in-depth on the subject.
Furthermore, it may be a chance to boost science to look into safer treatments for anxiety and similar disorders.
Graduated in Social Communication at the Federal University of Goiás. Passionate about digital media, pop culture, technology, politics and psychoanalysis.